Drug class
Betaine
Betaine is a specific active substance and metabolic agent rather than a broad therapeutic drug class. Read more about Betaine
5 drugs in this class 5 manufacturers
Drugs in Betaine
CYSTADANE (betaine)
BETAINE ANHYDROUS (betaine)
BETAINE ANHYDROUS (betaine)
BETAINE ANHYDROUS (betaine)
Betaine is a specific active substance and metabolic agent rather than a broad therapeutic drug class.
Pharmacologically, it functions as an organic methyl donor that remethylates homocysteine to methionine via the liver enzyme betaine-homocysteine methyltransferase.
Therapeutically, it is indicated to lower elevated blood and urine homocysteine concentrations in pediatric and adult patients with homocystinuria, including disorders arising from cystathionine beta-synthase deficiency, 5,10-methylenetetrahydrofolate reductase deficiency, or cobalamin cofactor metabolism defects.
By reducing toxic accumulations of homocysteine, it helps prevent serious complications such as thromboembolism, skeletal abnormalities, and neurological deterioration.
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fda.gov
Betaine is a specific active substance and metabolic agent rather than a broad therapeutic drug class
-
fda.gov
Betaine is a specific active substance and metabolic agent rather than a broad therapeutic drug class
-
nih.gov
Pharmacologically, it functions as an organic methyl donor that remethylates homocysteine to methionine via the liver enzyme betaine-homocysteine methyltransferase
-
patsnap.com
Pharmacologically, it functions as an organic methyl donor that remethylates homocysteine to methionine via the liver enzyme betaine-homocysteine methyltransferase
-
fda.gov
Therapeutically, it is indicated to lower elevated blood and urine homocysteine concentrations in pediatric and adult patients with homocystinuria, including disorders arising from cystathionine beta-synthase deficiency, 5,10-methylenetetrahydrofolate reductase deficiency, or cobalamin cofactor metabolism defects
-
nih.gov
Therapeutically, it is indicated to lower elevated blood and urine homocysteine concentrations in pediatric and adult patients with homocystinuria, including disorders arising from cystathionine beta-synthase deficiency, 5,10-methylenetetrahydrofolate reductase deficiency, or cobalamin cofactor metabolism defects By reducing toxic accumulations of homocysteine, it helps prevent serious complications such as thromboembolism, skeletal abnormalities, and neurological deterioration
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