Allergic rhinitis due to house dust mite

Allergic rhinitis due to house dust mite is an immunoglobulin E (IgE)–mediated, type I hypersensitivity disorder of the upper respiratory tract triggered by exposure to aeroallergens from house dust mites, predominantly *Dermatophagoides pteronyssinus* and *Dermatophagoides farinae*

Allergic rhinitis due to house dust mite is an immunoglobulin E (IgE)–mediated, type I hypersensitivity disorder of the upper respiratory tract triggered by exposure to aeroallergens from house dust mites, predominantly *Dermatophagoides pteronyssinus* and *Dermatophagoides farinae* Clinically, it typically presents as persistent or perennial rhinitis characterized by nasal congestion, rhinorrhea, paroxysmal sneezing, nasal and palatal pruritus, and postnasal drip, frequently associated with all...

Allergic rhinitis due to house dust mite is an immunoglobulin E (IgE)–mediated, type I hypersensitivity disorder of the upper respiratory tract triggered by exposure to aeroallergens from house dust mites, predominantly *Dermatophagoides pteronyssinus* and *Dermatophagoides farinae* Inhalation of microscopic mite fecal pellets and body fragments provokes allergen cross-linking on sensitized nasal mucosal mast cells, prompting the rapid release of histamine and other inflammatory mediators follow...

Inhalation of microscopic mite fecal pellets and body fragments provokes allergen cross-linking on sensitized nasal mucosal mast cells, prompting the rapid release of histamine and other inflammatory mediators followed by a late-phase influx of eosinophils and T-helper 2 cells Clinically, it typically presents as persistent or perennial rhinitis characterized by nasal congestion, rhinorrhea, paroxysmal sneezing, nasal and palatal pruritus, and postnasal drip, frequently associated with allergic ...

Inhalation of microscopic mite fecal pellets and body fragments provokes allergen cross-linking on sensitized nasal mucosal mast cells, prompting the rapid release of histamine and other inflammatory mediators followed by a late-phase influx of eosinophils and T-helper 2 cells

Clinically, it typically presents as persistent or perennial rhinitis characterized by nasal congestion, rhinorrhea, paroxysmal sneezing, nasal and palatal pruritus, and postnasal drip, frequently associated with allergic conjunctivitis or comorbid asthma