Indication

Arsenic toxicity

Arsenic toxicity is a potentially fatal clinical condition resulting from acute or chronic exposure to inorganic or organic arsenic compounds, most frequently via contaminated groundwater, occupational exposure, or intentional ingestion. Read more about Arsenic toxicity

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BAL IN OIL (dimercaprol)

GOLD TOXICITY·+3

AKORN INC.

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About Arsenic toxicity.

It impairs multisystem physiology primarily by binding to sulfhydryl groups on essential cellular enzymes, depleting glutathione, generating oxidative stress, and disrupting cellular respiration.

Acute poisoning presents prominently with severe hemorrhagic gastroenteritis, cardiovascular collapse, and cerebral edema, whereas chronic toxicity affects the integumentary, nervous, cardiovascular, and hematologic systems, manifesting as characteristic hyperpigmentation, palmoplantar hyperkeratosis, painful peripheral sensorimotor neuropathy, and an increased risk of internal and cutaneous malignancies.

nih.gov
Arsenic toxicity is a potentially fatal clinical condition resulting from acute or chronic exposure to inorganic or organic arsenic compounds, most frequently via contaminated groundwater, occupational exposure, or intentional ingestion
nih.gov
Arsenic toxicity is a potentially fatal clinical condition resulting from acute or chronic exposure to inorganic or organic arsenic compounds, most frequently via contaminated groundwater, occupational exposure, or intentional ingestion Acute poisoning presents prominently with severe hemorrhagic gastroenteritis, cardiovascular collapse, and cerebral edema, whereas chronic toxicity affects the integumentary, nervous, cardiovascular, and hematologic systems, manifesting as characteristic hyperpig...
nih.gov
Arsenic toxicity is a potentially fatal clinical condition resulting from acute or chronic exposure to inorganic or organic arsenic compounds, most frequently via contaminated groundwater, occupational exposure, or intentional ingestion
nih.gov
It impairs multisystem physiology primarily by binding to sulfhydryl groups on essential cellular enzymes, depleting glutathione, generating oxidative stress, and disrupting cellular respiration
nih.gov
It impairs multisystem physiology primarily by binding to sulfhydryl groups on essential cellular enzymes, depleting glutathione, generating oxidative stress, and disrupting cellular respiration
nih.gov
It impairs multisystem physiology primarily by binding to sulfhydryl groups on essential cellular enzymes, depleting glutathione, generating oxidative stress, and disrupting cellular respiration
nih.gov
Acute poisoning presents prominently with severe hemorrhagic gastroenteritis, cardiovascular collapse, and cerebral edema, whereas chronic toxicity affects the integumentary, nervous, cardiovascular, and hematologic systems, manifesting as characteristic hyperpigmentation, palmoplantar hyperkeratosis, painful peripheral sensorimotor neuropathy, and an increased risk of internal and cutaneous malignancies
cdc.gov
Acute poisoning presents prominently with severe hemorrhagic gastroenteritis, cardiovascular collapse, and cerebral edema, whereas chronic toxicity affects the integumentary, nervous, cardiovascular, and hematologic systems, manifesting as characteristic hyperpigmentation, palmoplantar hyperkeratosis, painful peripheral sensorimotor neuropathy, and an increased risk of internal and cutaneous malignancies
cdc.gov
Acute poisoning presents prominently with severe hemorrhagic gastroenteritis, cardiovascular collapse, and cerebral edema, whereas chronic toxicity affects the integumentary, nervous, cardiovascular, and hematologic systems, manifesting as characteristic hyperpigmentation, palmoplantar hyperkeratosis, painful peripheral sensorimotor neuropathy, and an increased risk of internal and cutaneous malignancies

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