Drug class
Inotropic Agents
Inotropic agents are a class of cardiovascular medications that alter the force or energy of myocardial contractions, with clinical usage predominantly referring to positive inotropes that augment cardiac contractility. Read more about Inotropic Agents
26 drugs in this class 17 manufacturers
Drugs in Inotropic Agents
MILRINONE LACTATE
DOBUTAMINE HCL-D5W (dobutamine HCl in dextrose 5 % in water)
MILRINONE IN 5% DEXTROSE (milrinone lactate/dextrose 5 % in water)
MILRINONE IN 5% DEXTROSE (milrinone lactate/dextrose 5 % in water)
MILRINONE LACTATE
DOBUTAMINE HCL
MILRINONE LACTATE
MILRINONE LACTATE
Inotropic agents are a class of cardiovascular medications that alter the force or energy of myocardial contractions, with clinical usage predominantly referring to positive inotropes that augment cardiac contractility.
These agents primarily work through distinct pharmacological mechanisms—such as beta-adrenergic receptor stimulation, phosphodiesterase enzyme inhibition, or modulation of intracellular calcium handling—to enhance stroke volume and cardiac output.
In clinical practice, they are primarily indicated for the management of acute decompensated heart failure, cardiogenic shock, and severe low-output states associated with cardiac surgery or refractory ventricular dysfunction to maintain systemic perfusion.
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nih.gov
Inotropic agents are a class of cardiovascular medications that alter the force or energy of myocardial contractions, with clinical usage predominantly referring to positive inotropes that augment cardiac contractility
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clevelandclinic.org
Inotropic agents are a class of cardiovascular medications that alter the force or energy of myocardial contractions, with clinical usage predominantly referring to positive inotropes that augment cardiac contractility
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drugs.com
Inotropic agents are a class of cardiovascular medications that alter the force or energy of myocardial contractions, with clinical usage predominantly referring to positive inotropes that augment cardiac contractility These agents primarily work through distinct pharmacological mechanisms—such as beta-adrenergic receptor stimulation, phosphodiesterase enzyme inhibition, or modulation of intracellular calcium handling—to enhance stroke volume and cardiac output In clinical practice, they are pri...
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fda.gov
These agents primarily work through distinct pharmacological mechanisms—such as beta-adrenergic receptor stimulation, phosphodiesterase enzyme inhibition, or modulation of intracellular calcium handling—to enhance stroke volume and cardiac output
-
nih.gov
These agents primarily work through distinct pharmacological mechanisms—such as beta-adrenergic receptor stimulation, phosphodiesterase enzyme inhibition, or modulation of intracellular calcium handling—to enhance stroke volume and cardiac output In clinical practice, they are primarily indicated for the management of acute decompensated heart failure, cardiogenic shock, and severe low-output states associated with cardiac surgery or refractory ventricular dysfunction to maintain systemic perfus...
-
nih.gov
These agents primarily work through distinct pharmacological mechanisms—such as beta-adrenergic receptor stimulation, phosphodiesterase enzyme inhibition, or modulation of intracellular calcium handling—to enhance stroke volume and cardiac output In clinical practice, they are primarily indicated for the management of acute decompensated heart failure, cardiogenic shock, and severe low-output states associated with cardiac surgery or refractory ventricular dysfunction to maintain systemic perfus...
-
nih.gov
In clinical practice, they are primarily indicated for the management of acute decompensated heart failure, cardiogenic shock, and severe low-output states associated with cardiac surgery or refractory ventricular dysfunction to maintain systemic perfusion
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