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Drug class

Ophthalmic Adrenergic Agents

Ophthalmic adrenergic agents are a therapeutic class of topical ocular medications that modulate adrenergic receptors—primarily through alpha-adrenergic receptor agonism (such as brimonidine or apraclonidine) or beta-adrenergic receptor blockade (such as timolol). Read more about Ophthalmic Adrenergic Agents

15 drugs in this class 14 manufacturers

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UPNEEQ (oxymetazoline HCl/PF)

OPHTHALMIC ADRENERGIC AGENTS · ACQUIRED BLEPHAROPTOSIS

RVL PHARMACEUTI

BRIMONIDINE TARTRATE-TIMOLOL (brimonidine tartrate/timolol maleate)

OPHTHALMIC ADRENERGIC AGENTS · OCULAR HYPERTENSION ·+1

PACIFIC-ZYDUS (

BRIMONIDINE TARTRATE-TIMOLOL (brimonidine tartrate/timolol maleate)

OPHTHALMIC ADRENERGIC AGENTS · OCULAR HYPERTENSION ·+1

SANDOZ

BRIMONIDINE TARTRATE-TIMOLOL (brimonidine tartrate/timolol maleate)

OPHTHALMIC ADRENERGIC AGENTS · OCULAR HYPERTENSION ·+1

CAPLIN STERILES

BRIMONIDINE TARTRATE-TIMOLOL (brimonidine tartrate/timolol maleate)

OPHTHALMIC ADRENERGIC AGENTS · OCULAR HYPERTENSION ·+1

LEADING PHARMA

BRIMONIDINE TARTRATE-TIMOLOL (brimonidine tartrate/timolol maleate)

OPHTHALMIC ADRENERGIC AGENTS · OCULAR HYPERTENSION ·+1

NORTHSTAR RX LL

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About Ophthalmic Adrenergic Agents.

Ophthalmic adrenergic agents are a therapeutic class of topical ocular medications that modulate adrenergic receptors—primarily through alpha-adrenergic receptor agonism (such as brimonidine or apraclonidine) or beta-adrenergic receptor blockade (such as timolol).

Their principal mechanism of action involves decreasing the production of aqueous humor in the ciliary body and, for certain alpha-agonists, enhancing uveoscleral outflow or inducing local vasoconstriction.

Clinically, these agents are primarily indicated for reducing intraocular pressure in the management of open-angle glaucoma and ocular hypertension, as well as for perioperative pressure spike prevention, pupil dilation, or ocular redness relief.

fda.gov
Ophthalmic adrenergic agents are a therapeutic class of topical ocular medications that modulate adrenergic receptors—primarily through alpha-adrenergic receptor agonism (such as brimonidine or apraclonidine) or beta-adrenergic receptor blockade (such as timolol) Their principal mechanism of action involves decreasing the production of aqueous humor in the ciliary body and, for certain alpha-agonists, enhancing uveoscleral outflow or inducing local vasoconstriction Clinically, these agents are p...
fda.gov
Ophthalmic adrenergic agents are a therapeutic class of topical ocular medications that modulate adrenergic receptors—primarily through alpha-adrenergic receptor agonism (such as brimonidine or apraclonidine) or beta-adrenergic receptor blockade (such as timolol)
medpagetoday.com
Ophthalmic adrenergic agents are a therapeutic class of topical ocular medications that modulate adrenergic receptors—primarily through alpha-adrenergic receptor agonism (such as brimonidine or apraclonidine) or beta-adrenergic receptor blockade (such as timolol) Their principal mechanism of action involves decreasing the production of aqueous humor in the ciliary body and, for certain alpha-agonists, enhancing uveoscleral outflow or inducing local vasoconstriction Clinically, these agents are p...
nih.gov
Ophthalmic adrenergic agents are a therapeutic class of topical ocular medications that modulate adrenergic receptors—primarily through alpha-adrenergic receptor agonism (such as brimonidine or apraclonidine) or beta-adrenergic receptor blockade (such as timolol)
nih.gov
Ophthalmic adrenergic agents are a therapeutic class of topical ocular medications that modulate adrenergic receptors—primarily through alpha-adrenergic receptor agonism (such as brimonidine or apraclonidine) or beta-adrenergic receptor blockade (such as timolol) Their principal mechanism of action involves decreasing the production of aqueous humor in the ciliary body and, for certain alpha-agonists, enhancing uveoscleral outflow or inducing local vasoconstriction Clinically, these agents are p...
nih.gov
Their principal mechanism of action involves decreasing the production of aqueous humor in the ciliary body and, for certain alpha-agonists, enhancing uveoscleral outflow or inducing local vasoconstriction
nih.gov
Their principal mechanism of action involves decreasing the production of aqueous humor in the ciliary body and, for certain alpha-agonists, enhancing uveoscleral outflow or inducing local vasoconstriction