Indication

Complicated skin and skin structure Streptococcus dysgalactiae infection

Complicated skin and skin structure Streptococcus dysgalactiae infection is a severe, deep-seated bacterial infection of the integumentary system involving the dermis, subcutaneous tissue, fascial planes, or underlying muscle. Read more about Complicated skin and skin structure Streptococcus dysgalactiae infection

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Common Complicated skin and skin structure Streptococcus dysgalactiae infection therapies.

Showing 10–18 of 43 drugs

DALVANCE (dalbavancin HCl)

AGENTS TO TREAT RESISTANT GRAM POSITIVE ORGANISMS·COMPLICATED SKIN AND SKIN STRUCTURE STAPHYLOCOCCUS AUREUS INFECTION·+6
DURATA/ALLERGAN

DAPTOMYCIN

AGENTS TO TREAT RESISTANT GRAM POSITIVE ORGANISMS·STAPHYLOCOCCUS AUREUS SEPTICEMIA·+6
Hospira

DAPTOMYCIN

AGENTS TO TREAT RESISTANT GRAM POSITIVE ORGANISMS·STAPHYLOCOCCUS AUREUS SEPTICEMIA·+6
XELLIA PHARMACE

DAPTOMYCIN

AGENTS TO TREAT RESISTANT GRAM POSITIVE ORGANISMS·STAPHYLOCOCCUS AUREUS SEPTICEMIA·+6
MYLAN INSTITUTI

DAPTOMYCIN

AGENTS TO TREAT RESISTANT GRAM POSITIVE ORGANISMS·STAPHYLOCOCCUS AUREUS SEPTICEMIA·+6
NORTHSTAR RX LL

DAPTOMYCIN

AGENTS TO TREAT RESISTANT GRAM POSITIVE ORGANISMS·STAPHYLOCOCCUS AUREUS SEPTICEMIA·+6
DR.REDDY'S LAB

DAPTOMYCIN

AGENTS TO TREAT RESISTANT GRAM POSITIVE ORGANISMS·STAPHYLOCOCCUS AUREUS SEPTICEMIA·+6
BLUEPOINT LABOR

DAPTOMYCIN

STAPHYLOCOCCUS AUREUS SEPTICEMIA·+6
CIPLA USA, INC.

DAPTOMYCIN

AGENTS TO TREAT RESISTANT GRAM POSITIVE ORGANISMS·STAPHYLOCOCCUS AUREUS SEPTICEMIA·+6
CIVICA, INC.
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About Complicated skin and skin structure Streptococcus dysgalactiae infection.

Complicated skin and skin structure Streptococcus dysgalactiae infection is a severe, deep-seated bacterial infection of the integumentary system involving the dermis, subcutaneous tissue, fascial planes, or underlying muscle.

Primarily caused by the beta-hemolytic Lancefield group C or G pathogen Streptococcus dysgalactiae (predominantly subspecies equisimilis), these infections frequently arise in patients with underlying systemic comorbidities, cutaneous breaches, or impaired lymphatic drainage.

Pathophysiologically, the bacterium invades deep tissue spaces via extracellular virulence factors and toxins, triggering intense localized inflammation, tissue necrosis, or abscess formation, often presenting as extensive cellulitis, surgical wound infections, or necrotizing fasciitis accompanied by systemic signs of toxicity.

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