Indication
Metastatic castration-sensitive prostate cancer
Metastatic castration-sensitive prostate cancer is an advanced stage of malignancy originating in the male prostate gland that has disseminated beyond regional pelvic lymph nodes to distant sites, most commonly the skeleton, non-regional lymph nodes, or visceral organs. Read more about Metastatic castration-sensitive prostate cancer
31 drugs 28 manufacturers
Drugs that treat it
Common Metastatic castration-sensitive prostate cancer therapies.
ABIRATERONE ACETATE
ABIRATERONE ACETATE
ABIRATERONE ACETATE
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Coming soonMetastatic castration-sensitive prostate cancer is an advanced stage of malignancy originating in the male prostate gland that has disseminated beyond regional pelvic lymph nodes to distant sites, most commonly the skeleton, non-regional lymph nodes, or visceral organs.
The defining pathophysiologic feature of this clinical state is the continued dependence of the metastatic tumor cells on androgen receptor signaling pathways for survival and proliferation.
Consequently, patients remain functionally responsive to androgen suppression, whether they are treatment-naive or previously exposed to medical or surgical castration without having developed disease progression at castrate levels of serum testosterone.
- nih.gov
Metastatic castration-sensitive prostate cancer is an advanced stage of malignancy originating in the male prostate gland that has disseminated beyond regional pelvic lymph nodes to distant sites, most commonly the skeleton, non-regional lymph nodes, or visceral organs
- mayoclinic.org
Metastatic castration-sensitive prostate cancer is an advanced stage of malignancy originating in the male prostate gland that has disseminated beyond regional pelvic lymph nodes to distant sites, most commonly the skeleton, non-regional lymph nodes, or visceral organs Consequently, patients remain functionally responsive to androgen suppression, whether they are treatment-naive or previously exposed to medical or surgical castration without having developed disease progression at castrate level...
- nih.gov
The defining pathophysiologic feature of this clinical state is the continued dependence of the metastatic tumor cells on androgen receptor signaling pathways for survival and proliferation
- nih.gov
Consequently, patients remain functionally responsive to androgen suppression, whether they are treatment-naive or previously exposed to medical or surgical castration without having developed disease progression at castrate levels of serum testosterone
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