Indication

Thrombosis prevention during percutaneous coronary intervention in heparin-induced thrombocytopenia

Thrombosis prevention during percutaneous coronary intervention in heparin-induced thrombocytopenia refers to the essential procedural anticoagulation strategy utilized in adult cardiovascular patients undergoing transcatheter coronary revascularization who have active or previous immune-mediated heparin-induced thrombocytopenia. Read more about Thrombosis prevention during percutaneous coronary intervention in heparin-induced thrombocytopenia

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Common Thrombosis prevention during percutaneous coronary intervention in heparin-induced thrombocytopenia therapies.

Showing 28–36 of 49 drugs

ARGATROBAN

THROMBIN INHIBITORS (NON-HEPARINOID)·THROMBOSIS IN HEPARIN-INDUCED THROMBOCYTOPENIA·+2
Hikma Pharmaceuticals USA

BIVALIRUDIN

THROMBIN INHIBITORS (NON-HEPARINOID)·THROMBOSIS WITH HEPARIN-INDUCED THROMBOCYTOPENIA DURING PERCUTANEOUS CORONARY INTERVENTION·+2
MYLAN INSTITUTI

ANGIOMAX (bivalirudin)

THROMBOSIS WITH HEPARIN-INDUCED THROMBOCYTOPENIA DURING PERCUTANEOUS CORONARY INTERVENTION·+2
MEDICINE/SANDOZ

ANGIOMAX (bivalirudin)

THROMBIN INHIBITORS (NON-HEPARINOID)·THROMBOSIS WITH HEPARIN-INDUCED THROMBOCYTOPENIA DURING PERCUTANEOUS CORONARY INTERVENTION·+2
SANDOZ

BIVALIRUDIN

THROMBIN INHIBITORS (NON-HEPARINOID)·THROMBOSIS WITH HEPARIN-INDUCED THROMBOCYTOPENIA DURING PERCUTANEOUS CORONARY INTERVENTION·+2
PAR HEALTH USA,
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About Thrombosis prevention during percutaneous coronary intervention in heparin-induced thrombocytopenia.

Thrombosis prevention during percutaneous coronary intervention in heparin-induced thrombocytopenia refers to the essential procedural anticoagulation strategy utilized in adult cardiovascular patients undergoing transcatheter coronary revascularization who have active or previous immune-mediated heparin-induced thrombocytopenia.

Heparin-induced thrombocytopenia is a severe, antibody-driven hypercoagulable state triggered by antibodies against platelet factor 4–heparin complexes, which causes intravascular platelet activation, extensive thrombin generation, and a paradoxical predisposition to limb- and life-threatening arterial and venous clotting.

In this clinical setting, standard heparin re-exposure is contraindicated, yet mechanical vascular injury and coronary stent placement create a potent thrombogenic stimulus that requires alternative, non-heparin anticoagulation to prevent acute catheter-induced clotting, intraprocedural myocardial infarction, and stent thrombosis.

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