Indication
Zinc deficiency
Zinc deficiency is a systemic nutritional disorder characterized by an insufficient biological supply or impaired absorption of the essential trace element zinc. Read more about Zinc deficiency
36 drugs 27 manufacturers
Drugs that treat it
Common Zinc deficiency therapies.
ZINC (zinc amino acid chelate)
ZINC
ZINC (zinc gluconate)
ZINC SULFATE
VITAMELTS FAST DISSOLVE (zinc oxide)
ZINC CHELATED (zinc amino acid chelate)
ZINC CHLORIDE
ZINC CHELATED (zinc amino acid chelate)
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Coming soonZinc deficiency is a systemic nutritional disorder characterized by an insufficient biological supply or impaired absorption of the essential trace element zinc.
Because zinc serves as an essential structural and catalytic cofactor for hundreds of metalloenzymes and transcription factors, its deficiency impairs cellular division, protein synthesis, and antioxidant defense across multiple organ systems.
Clinically, it frequently affects rapidly proliferating tissues, manifesting in pediatric and adult populations as impaired cell-mediated immunity, impaired wound healing, alopecia, dysgeusia, and classic periorificial or acral dermatitis.
In infants and children, severe or chronic deficiency leads to growth failure, delayed sexual maturation, diarrhea, and heightened susceptibility to severe systemic infections.
- nih.gov
Zinc deficiency is a systemic nutritional disorder characterized by an insufficient biological supply or impaired absorption of the essential trace element zinc Because zinc serves as an essential structural and catalytic cofactor for hundreds of metalloenzymes and transcription factors, its deficiency impairs cellular division, protein synthesis, and antioxidant defense across multiple organ systems Clinically, it frequently affects rapidly proliferating tissues, manifesting in pediatric and ad...
- nih.gov
Zinc deficiency is a systemic nutritional disorder characterized by an insufficient biological supply or impaired absorption of the essential trace element zinc
- nih.gov
Because zinc serves as an essential structural and catalytic cofactor for hundreds of metalloenzymes and transcription factors, its deficiency impairs cellular division, protein synthesis, and antioxidant defense across multiple organ systems Clinically, it frequently affects rapidly proliferating tissues, manifesting in pediatric and adult populations as impaired cell-mediated immunity, impaired wound healing, alopecia, dysgeusia, and classic periorificial or acral dermatitis
- nih.gov
Clinically, it frequently affects rapidly proliferating tissues, manifesting in pediatric and adult populations as impaired cell-mediated immunity, impaired wound healing, alopecia, dysgeusia, and classic periorificial or acral dermatitis In infants and children, severe or chronic deficiency leads to growth failure, delayed sexual maturation, diarrhea, and heightened susceptibility to severe systemic infections
- nih.gov
Clinically, it frequently affects rapidly proliferating tissues, manifesting in pediatric and adult populations as impaired cell-mediated immunity, impaired wound healing, alopecia, dysgeusia, and classic periorificial or acral dermatitis In infants and children, severe or chronic deficiency leads to growth failure, delayed sexual maturation, diarrhea, and heightened susceptibility to severe systemic infections
- medlineplus.gov
Clinically, it frequently affects rapidly proliferating tissues, manifesting in pediatric and adult populations as impaired cell-mediated immunity, impaired wound healing, alopecia, dysgeusia, and classic periorificial or acral dermatitis In infants and children, severe or chronic deficiency leads to growth failure, delayed sexual maturation, diarrhea, and heightened susceptibility to severe systemic infections
- nih.gov
In infants and children, severe or chronic deficiency leads to growth failure, delayed sexual maturation, diarrhea, and heightened susceptibility to severe systemic infections
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